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Why your resting heart rate changes

Explore your usual pulse, everyday changes, and when to seek care.

Key Takeaways

  • The general adult resting heart rate range is 60–100 beats per minute. The 45–85 range used in Japanese comprehensive health checkups is a screening reference interval for measurements taken lying down, not an interchangeable definition of normal.[1][2][3]
  • Heart rate naturally changes with stress, fever, exercise, and sleep. The context and persistence of a change matter.[1][6]
  • Comparing measurements taken under similar conditions with your own usual pulse can be useful. A personal baseline cannot diagnose or rule out disease.[4][5]
  • Seek medical advice for a persistent resting pulse above 100 or below 50, or an irregular rhythm. For chest pain, severe breathlessness, fainting, or other emergency symptoms, call your local emergency number now; do not wait for a pulse threshold.[1][5]
Contents

This is general educational information, not a substitute for medical diagnosis or treatment. A personal baseline, online measurement, or app reading cannot diagnose or rule out disease. Online measurement estimates pulse rate and is not an ECG. Consult a healthcare professional about concerning changes.

A blood pressure monitor or smartwatch shows your heart rate: 62 in the morning, 78 later in the day, higher when you have a cold. What is that number telling you? Starting with how the heart works, this article follows the reasons its pace changes.

A number alone cannot tell you whether you are healthy. Through a fictional year in the life of Misaki, we explore your usual pulse alongside the reason for a change, how long it lasts, its rhythm, and any symptoms. Numbers in square brackets refer to the sources below.

Prologue: The Same Heart, Different Numbers

A pulse of 62 on waking and over 110 after running up stairs can both reflect what your body needs at that moment. Your heart is designed to change its pace.[6]

THE MECHANISM, EXPLAINED

Changing demand. A changing response.

SituationDemand → response
  1. At rest

    Less demand from movementA resting pace

  2. During activity

    Working muscles need more oxygenCardiac output rises; pulse usually increases

  3. In recovery

    Demand settles after activityPulse moves back toward its resting pace

A qualitative sequence, not target rates or recovery times. Responses vary; recovery alone cannot rule out disease. [1]

Arrows show functional relationships. This is not an individual diagnostic result.

It is 6:30 a.m. Misaki, a 36-year-old office worker, turns off her alarm and stays still in bed for a while. Her pulse reading that morning is 62 beats per minute.

An hour later, she runs up the station stairs to catch her train. Her heart pounds. A reading afterward is more than 110 beats per minute. The same person, the same heart, yet a very different number.

“Which one is my real heart rate?” Both are real. Nerves and hormones continually adjust how fast and how forcefully the heart contracts. When the body needs more blood, its pace rises; when the body rests, it slows. These changes can happen without illness.[6]

Who sets that pace, and how? Which changes are expected, and which deserve medical attention? Let us look inside the heart while following a year in Misaki’s life.

Chapter 1: Your Heart Is Two Pumps Working Together

The right side of the heart pumps blood to the lungs; the left side pumps blood returning from the lungs to the body. Four chambers work together, with valves preventing backward flow.[6][7]

A WORKING CIRCUIT, NOT A HEART PICTURE

Two pumps. One closed circuit.

The right side sends blood to the lungs. The left side sends it to the body. Follow the moving flow.
LUNGS
Pick up oxygen
RIGHT PUMPRight atrium → ventricle
LEFT PUMPLeft atrium → ventricle
ONE CIRCULATIONWorking togetherNot alternating left and right
BODY TISSUES
Deliver oxygen
↑↓
01 / 04
Body → right pump

After delivering oxygen to tissues, blood returns to the right atrium.

Lower-oxygen bloodOxygen-rich blood
A functional model of flow and oxygen exchange. Blue is not literal blood color. Motion does not represent measured speed, pressure, or heart rate; the two pumps work in coordination. [7]

The heart has four chambers. The two upper chambers are the atria, and the two lower chambers are the ventricles. The atria receive returning blood and pass it into the ventricles, which pump it out of the heart.[7]

These chambers do not work independently. Think of two pumps side by side, each with a receiving chamber and a sending chamber. The right atrium and right ventricle receive blood returning from the body and send it to the lungs, where it picks up oxygen and releases carbon dioxide. The left atrium and left ventricle receive that oxygen-rich blood and send it around the body, delivering oxygen and nutrients to tissues.[7]

The two pumps act in coordination. First, the atria contract to help fill the ventricles. Then the ventricles contract, sending blood to the lungs and the rest of the body. A brief delay as the electrical signal travels from the atria toward the ventricles allows time for filling.[6][7]

LOOK CLOSER

Valves switch the route. Blood moves one way.

INLET: openOUTLET: closed

Watch the valves: inlet and outlet are not open together. This is a functional model of ventricular filling and ejection.

1 / 4
Relaxation begins

The inlet valve opens while the outlet stays closed.

Shapes, movement, and timing are exaggerated for explanation. Not measured data or an individual diagnostic result. [7]

Valves at the entrance and exit of each ventricle keep blood moving in one direction, like nonreturn valves in a pump. The two main heart sounds heard through the chest are associated with valves closing; they are not simply the sounds of the atria and ventricles contracting in turn.[7]

At 70 beats per minute, the heart beats 4,200 times an hour and about 100,000 times a day. This cycle of contraction and relaxation continues while you sleep.

Chapter 2: Who Sets the Rhythm? Your Natural Pacemaker

A normal heartbeat starts in the sinus node in the right atrium. The vagus nerve acts like a brake, sympathetic nerves act like an accelerator, and hormones also help adjust the pace.[6][8]

THE MECHANISM, EXPLAINED

The signal sequence. The controls on its pace.

  1. Sinus node

    An electrical signal begins

  2. Atria

    Contract to move blood into the ventricles

  3. AV node

    Briefly delays the signal before passing it on

  4. Ventricles

    Contract to pump blood to the lungs and body

Vagus nerveActs to slow the sinus node’s pace

Sympathetic nervesAct to increase the sinus node’s pace

Above: signal order. Below: pace regulation. Motion illustrates sequence, not real conduction timing. [6] [8]

Arrows show functional relationships. This is not an individual diagnostic result.

Near the top of the right atrium is a small group of cells called the sinus node, or sinoatrial node. This is the heart’s natural pacemaker. In normal sinus rhythm, its electrical signals travel through the atria toward the ventricles and trigger each heartbeat. If conduction is abnormal or signals arise elsewhere, the number of sinus-node signals may not match the heart’s actual beats.[2][6]

LOOK CLOSER

An electrical signal starts an ordered contraction

  1. 1Sinus node
  2. 2Atria
  3. 3AV node
  4. 4Ventricles

Notice the pause at the AV node before the signal reaches the ventricles. Symbols and timing are explanatory, not measured electrical recordings.

1 / 4
Sinus node

A normal heartbeat’s electrical signal starts here.

Shapes, movement, and timing are exaggerated for explanation. Not measured data or an individual diagnostic result. [6] [7]

Without autonomic nervous system input, the sinus node’s intrinsic pace is often described as roughly 100–110 signals per minute. Resting heart rate is commonly lower because the vagus nerve, which connects the brain and heart, normally applies a gentle braking effect.[8]

When you begin moving, that brake is released, and sympathetic nerves add an accelerating influence. Hormones also adjust heart rate and the strength of contraction. These were some of the adjustments taking place when Misaki ran up the station stairs.[6][8]

The accelerator-and-brake analogy will return in later chapters. Emotional stress, fever, and training all influence the systems that regulate the heart’s pace.

Are Heart Rate and Pulse Rate the Same?

Heart rate counts the heart’s beats over a given time. Pulse rate counts the pressure waves you can feel in an artery, such as at your wrist. Usually the two match. With some arrhythmias, however, a heartbeat does not produce a strong enough pulse to be felt at the wrist, so the pulse count can be lower.[5]

LOOK CLOSER

A pulse is a pressure wave travelling along an artery

Watch the wall expansion travel to the right. Fixed dots are reference markers to distinguish the wave from blood movement, not a claim that blood stands still.

1 / 4
A beat creates a pressure change

The pressure wave begins to travel into the artery.

Shapes, movement, and timing are exaggerated for explanation. Not measured data or an individual diagnostic result. [5] [7]

A Faster Pulse When Breathing In Can Be Normal

Even at rest, the pulse may become slightly faster as you breathe in and slower as you breathe out. This normal variation is called respiratory sinus arrhythmia and is especially common in healthy children and young adults. Despite its name, it is usually not a disease. A new irregular pulse should not, however, be assumed to be this harmless variation based on a sensation or pulse number alone.[9]

Chapter 3: What Is a Normal Resting Heart Rate?

The general adult resting heart rate range is 60–100 beats per minute. The Japanese checkup interval of 45–85 applies to screening measurements taken lying down and serves a different purpose.[1][2][3]

THE MECHANISM, EXPLAINED

Make the conditions comparable first

Similar time

When rested and settled

Same posture

Reduce position-related differences

Same method

Keep device and technique consistent

Compare with your usual readings

Record sleep, illness, medicines, and symptoms alongside the reading

Interpret change with context and symptoms. A familiar number does not rule out illness.

Measurement basics [1] · Within-person variability [4]

Arrows show functional relationships. This is not an individual diagnostic result.

Resting heart rate is your heart rate while sitting or lying quietly and feeling settled. The usual adult reference is 60–100 beats per minute, but a rate below 60 can be normal in younger people and those who exercise regularly. Symptoms, medications, fitness, and measurement conditions all affect interpretation.[1][2]

The Japan Society of Ningen Dock and Preventive Medical Care uses 45–85 beats per minute as a reference interval for measurements taken lying down. Associations between heart rate and future cardiovascular risk inform its upper limit. A Japan Heart Foundation booklet describes an average of 65 ± 10 in health-checkup participants. These figures relate to particular screening settings and populations. They neither replace the general adult range nor mean that a result outside the interval automatically indicates disease.[3][5]

There Is More Than One “Usual” Pulse

A study of wearable data from 92,457 people found an overall average resting heart rate of about 65. Individuals’ average values ranged from approximately 40 to 109. The study had no clinical data to establish participants’ health, so that observed spread is not a healthy reference interval. A wearable’s calculated daily resting heart rate also differs from a single seated manual pulse measurement.[4]

Within an individual, readings were much more stable. The median measure of within-week variation was about three beats, and around 80% of participants had a difference of less than 10 beats even in their most variable week. These research observations are not personal thresholds for deciding when medical attention is needed.[4]

A reading of 70 may be ordinary for one person and higher than usual for another. Just as you have a usual body temperature, you can have a usual pulse. A baseline can help you notice a change, but cannot diagnose a condition or show that you are disease-free simply because the number is familiar.

Age and Sex Make Some Difference

In the same study, average resting heart rate rose until around age 50 and then declined, and women had higher averages than men across age groups. Lower averages were associated with 7–7.5 hours of sleep, while being underweight or having obesity was associated with higher averages. Yet age, sex, body size, and sleep together explained only about one tenth of the differences between people. These are observational associations, not proof that a particular sleep duration will lower anyone’s pulse.[4]

Prepare for Repeatable Online Measurements

Resting readings are easier to compare when taken at a similar time, in the same position, and under similar conditions. HeartScan uses your phone’s accelerometer to detect tiny chest vibrations using seismocardiography (SCG), not a camera. Recline comfortably and follow the tool’s guidance for starting and placing your phone on your chest. A heart rate estimate is not the same as an ECG assessment of heart rhythm.

For two weeks, Misaki checks her readings while seated after her morning wash. They fall between 60 and 64, giving her a usual value of about 62. A reading in bed is not taken under exactly the same conditions, so she uses the seated routine for future comparisons.

  1. Avoid measuring immediately after exercise. Rest for about five minutes, and recline comfortably for your HeartScan measurement.
  2. Use a similar time and posture for comparisons, and consider recent activity, stress, fever, and medications when interpreting readings.[1][5]
  3. Open the HeartScan online tool and follow its guidance for starting, placing your phone on your chest, and remaining still.
  4. If you want to compare readings over time, make your own note of the displayed pulse rate, date, time, posture, and how you feel. Seek medical advice for persistent unexplained changes or an irregular pulse.[1][5]

Chapter 4: Why Is My Heart Rate High When I Am Sitting Still?

Stress, pain, heat, and some medications can raise heart rate even without physical activity. A persistent, unexplained increase needs medical assessment rather than an assumption that it is stress.[1][10]

THE MECHANISM, EXPLAINED

Follow the cause, not just the pulse

StressHeatStimulants, including caffeine
Autonomic and physiological responses

The pathway and strength of response depend on the trigger

Pulse may become faster

[1] [8]

Arrows show functional relationships. This is not an individual diagnostic result.

One afternoon, Misaki sits in a meeting room waiting to present to senior executives. She is not moving, but her pulse is 92, a full 30 beats above her usual 62.

The accelerator can work even when your muscles are still. Anxiety, excitement, sadness, and pain can all affect heart rate. In Misaki’s story, her pulse returns to its usual level about an hour after the presentation. A fast pulse alone, however, cannot establish that nervousness is the cause.[1]

Things That Can Temporarily Raise Heart Rate

Sleep and season can also be associated with resting heart rate. In the study of about 90,000 people, average rates were lowest among those sleeping 7–7.5 hours. Across the study population, rates were highest in early January and lowest in late July, a difference of about two beats. These group patterns do not mean every person follows the same seasonal rhythm.[4]

Other medicines can slow the heart, including beta blockers and certain calcium channel blockers. Medication is important context when interpreting your usual pulse. Do not stop a prescribed medicine or change its dose on your own.[1]

  • Emotional or physical stress, including anxiety and pain.[1][2]
  • A brief increase just after standing up.[1]
  • Hot conditions or a rise in body temperature.[1]
  • Caffeine, alcohol, and smoking; individual responses vary.[2]
  • Some cold and allergy medicines containing stimulants such as pseudoephedrine.[11]
  • Fever and infection.[10]

When the Increase Persists

An unexplained high resting heart rate can be associated with conditions such as anemia, respiratory disease, an overactive thyroid, or heart failure. A pulse number cannot distinguish these causes.[10]

The reason for a change and how long it lasts matter as much as the number. A temporary rise that resolves when its trigger passes differs from an unexplained, sustained change. A rhythm that suddenly starts and stops, or becomes irregular, raises a different set of questions covered in Chapter 7.

  • If there is an apparent trigger such as nervousness, the pulse returns to normal with rest, and there are no other symptoms, recording the pattern can be useful.[10]
  • Seek medical advice if readings remain higher than usual without a clear reason, or resting heart rate stays above 100. An ECG may be needed.[5][10]
  • For chest pain, severe breathlessness, fainting, marked dizziness, or palpitations with a cold sweat, call your local emergency number now. Do not assume anxiety or wait for the pulse to exceed 100.[1][5]

Chapter 5: Why Does a Cold Make My Pulse Faster?

Fever and infection can raise heart rate, but temperature cannot precisely predict an individual’s pulse. A wearable reading alone cannot diagnose an infection.[1][12][14]

THE MECHANISM, EXPLAINED

Why the pulse often rises during fever

Fever / infectionHigher physiological demandHeart rate often rises

Observed in research: 27 young men (1986)

Mean after recovery
66.5 bpm
Mean during fever
84 bpm

Mean temperature during fever: 38.4°C. These are group averages from a small, restricted study, not a formula for your pulse or a safety threshold. Source: Karjalainen & Viitasalo [12]

Mechanism above; published study averages below. Not a personal prediction.

One December morning, Misaki wakes with a sore throat. Her temperature is 37.8°C, and her pulse is 73, eleven beats above her usual value despite resting. These are illustrative numbers in a fictional story, not a pairing everyone should expect.

Fever and Pulse Are Related, but Not by a Personal Formula

A small Finnish study compared ECG recordings from 27 young men during a febrile illness and after recovery. During fever, their average temperature was 38.4°C and their average heart rate was 84; after recovery, average heart rate was 66.5. Within that study, each 1°C increase was associated with an average increase of about 8.5 beats per minute, and heart rate also remained elevated during sleep.[12]

That average from a small group of young men is not a reliable formula for predicting your own heart rate. Medicines, the illness itself, and your usual pulse can also affect the result. Misaki’s reading of 73 should not be declared safe because it appears to fit a calculation based on the study.[1][12]

A Small Signal of Illness, Not a Diagnosis

In the United States, resting heart rate and sleep data from more than 47,000 wearable users improved estimates of state-level influenza-like illness activity. This was population surveillance, not a way to diagnose influenza in an individual.[13]

In a study of 32 people with COVID-19, 26 showed an increase in resting heart rate; changes were detected as early as 10 days before symptoms in some participants. This was a small study. A smartwatch cannot establish infection, and an unchanged reading cannot rule it out. Knowing your usual pulse may help you notice that something has changed, rather than tell you exactly what is wrong.[14]

Your Pulse May Take Time to Return After Illness

In data from 234 people with COVID-19, resting heart rate returned to its previous level an average of 79 days after symptom onset, later than step counts or sleep duration. About 14% still had a resting rate more than five beats above baseline beyond 133 days. These results describe that study group; they are neither an individual recovery timetable nor a period during which it is necessarily safe to wait.[15]

Recovery can continue after a fever ends. Persistent or worsening symptoms and heart rate changes deserve medical advice rather than an assumption that they are simply part of recovery.

What If There Is No Fever?

Possible contributors include changes before symptoms appear or during recovery, stimulant ingredients in some cold medicines, and altered sleep while unwell. The pulse alone cannot identify which explanation applies.[4][11][14][15]

Seek medical advice if resting heart rate stays above 100. If there is chest pain, severe breathlessness, fainting, or marked dizziness, call your local emergency number now rather than blaming a cold. Emergency symptoms matter regardless of the pulse count.[1][5]

In Misaki’s story, her fever ends after three days, but her morning pulse stays around 66–68 for a little longer. Feeling not quite back to normal, she eases her schedule and goes to bed earlier. This fictional course is not a rule that the same readings make medical care unnecessary.

Chapter 6: Why Regular Exercise Can Lower Resting Heart Rate

Regular exercise can lower resting heart rate, but the amount varies. A lower number is not always healthier, and a slow pulse with symptoms needs assessment.[1][16]

THE MECHANISM, EXPLAINED

Blood flow depends on more than heart rate

Stroke volumeBlood pumped per beat
multiplied by
Heart rateBeats per minute
equals
Cardiac outputBlood pumped per minute

Endurance adaptation can increase stroke volume, allowing the same resting blood flow with fewer beats.

A simplified physiological relationship. Training responses vary; a lower rate alone does not establish fitness or health. [7] [16]

Arrows show functional relationships. This is not an individual diagnostic result.

After the New Year, Misaki begins jogging in a nearby park three times a week. At first, she is out of breath after 15 minutes. By spring, she can run for 30. One morning, she notices that her usual pulse is now around 58 rather than 62.

A meta-analysis involving 12,952 people examined exercise and resting heart rate. Its abstract describes 191 studies; the results describe 181 articles after removing 10 duplicate publications. The table shows within-group changes before and after the interventions, not control-adjusted treatment effects. It cannot rank exercise types head to head or predict an individual response. The reduction with strength training was not statistically significant overall.[16]

Chapter 6: Why Regular Exercise Can Lower Resting Heart Rate
Exercise TypeChange in Exercise GroupsChange in Nonexercise Groups
Yoga−7.2%−2.0%
Endurance exercise, such as jogging or cycling−6.0%−0.1%
Strength training−2.5%+0.1%

How Much Might It Change?

The analysis found larger reductions among people with higher starting heart rates and smaller effects at older ages. Misaki’s change from 62 to 58 is similar in size to the average percentage change in endurance groups, but a study average cannot promise an individual result. Nor does an unchanged resting pulse mean exercise has no value.[16]

What Changes Inside the Heart?

Training can help the heart pump blood more efficiently, allowing it to meet resting needs with fewer beats. Changes in autonomic regulation can also contribute.[1][8]

LOOK CLOSER

Same total flow. Different amounts per beat.

A model delivering the same total over the same comparison period

Blood to sendTotal delivered
Smaller stroke volumeSmaller parcels, more often
Larger stroke volumeLarger parcels, fewer times

Same final level. Same total volume.Stroke volume and heart rate together determine the volume pumped over a given time.

Parcels, reservoirs, and movement are explanatory; blood does not flow in literal parcels. These are not measurements or targets and do not show actual stroke volume or heart rate. A lower heart rate is not always better; bodily demand and symptoms matter.

1 / 4
Compare the same total volume

One model sends smaller amounts; the other sends larger amounts per beat.

Shapes, movement, and timing are exaggerated for explanation. Not measured data or an individual diagnostic result. [7] [16]

Animal research suggests the sinus node itself may change too. Trained mice retained a slower heart rate even when autonomic influences were blocked with drugs. They also had reduced HCN4, an ion channel involved in pacemaker activity. This experiment does not, by itself, fully explain training effects in humans.[17]

Clinicians also consider how heart rate falls after exercise. Heart rate recovery depends on exercise intensity, whether recovery involves walking or complete rest, body position, medications, and the time of measurement. There is no single one-minute drop that everyone should use to declare their recovery normal. Seek advice if your heart rate does not rise much with exercise or if exercise brings unusual symptoms.[10]

Lower Is Not Always Better

Highly trained athletes can have very low resting heart rates. Athlete ECG criteria can regard sinus rates of at least 30 as a normal finding under specific conditions, including no symptoms and an appropriate rise with activity. This is not permission for the general public to wait until their pulse reaches 30 before seeking advice.[18]

Long-term, high-intensity endurance sport has separate considerations. A study of 52,755 Swedish long-distance cross-country skiers found an approximately 30% higher arrhythmia risk associated with completing at least five races over 10 years or racing faster. This observational finding does not show that jogging several times a week is dangerous.[19]

  • A slow pulse with fatigue, breathlessness, or dizziness needs medical advice. Fainting, severe breathlessness, or chest pain requires a call to your local emergency number now.[1][5]
  • Heart-rate-lowering medicines and strong vagal influences can also cause a low pulse; exercise is not the only explanation.[1][2]
  • Even athletes may need assessment for profound slowing or long pauses. Athlete criteria use rates of 30 or below or pauses of at least three seconds as prompts for evaluation, but symptoms must not be ignored while waiting for those numbers.[18]

A Closer Look: Do You Get Two Billion Heartbeats in a Lifetime?

There is no established fixed allowance of heartbeats that you can use up. An association between heart rate and lifespan does not prove that simply slowing the heart makes someone live longer.[21][23]

THE MECHANISM, EXPLAINED

A measure of state, not a countdown of life

A STATE METRIC

A clue to your current state

Responds to rest, activity, illness, and other influences

≠

NOT A FIXED BUDGET

A stock of lifetime beats to use up

Your remaining lifespan cannot be calculated from your pulse

Population associations are not an individual lifespan equation. A pulse rise during exercise is not evidence of spending a finite supply of life. [21] [23]

Arrows show functional relationships. This is not an individual diagnostic result.

You may have heard the claim that people with faster hearts die earlier. In Japan, this is often linked to Tatsuo Motokawa’s book Zo no Jikan, Nezumi no Jikan, or Elephant Time, Mouse Time. The idea is that animals with very different lifespans may have roughly similar lifetime heartbeat counts. Teaching material quoting the book describes about two billion beats: different animals living their time at different speeds. It is a compelling image.[20]

But does a person really have a fixed amount of time that ends when the final beat is spent?

The Number Depends on the Source

One analysis of mammals reported an average lifetime total of about 730 million heartbeats. Other writing describes about 800 million. Species differ, and sources use different estimates. Two billion should not be treated as a precise universal law; it is better read as an illustration of a broad relationship across species.[21][22]

Suppose we applied two billion to a person. At 70 beats per minute, the heart beats about 37 million times a year and would reach two billion in approximately 54 years. Average life expectancy in Japan is far longer. This arithmetic does not determine a human lifespan.

A Gauge, Not a Remaining Balance

Heart rate can be understood as a marker of physiological processes such as metabolism. That does not mean making the heart beat more slowly, by itself, extends life.[21]

In humans, a meta-analysis of 46 studies involving about 1.25 million people found that each 10-beat higher resting heart rate was associated with a 9% higher relative risk of death from any cause and an 8% higher relative risk of cardiovascular death. A group with rates above 80 had a 45% higher relative risk of all-cause death than the lowest-rate comparison group. These are population-level relative associations, not a calculation of an individual’s probability of dying or remaining years.[23]

Such risk associations also provide context for Japanese screening reference intervals. They do not create an immediate danger line at 85 beats per minute. Fitness, stress, sleep, and disease can all influence heart rate. Association is not the same as causation, and you should not change medication yourself to chase a lower number.[3][23]

Your heartbeat is not a shrinking balance of beats left to spend. It is more useful to think of it as a gauge that offers clues about your current condition.

Chapter 7: Skipped or Irregular Beats: What Is an Arrhythmia?

An arrhythmia is an abnormality of the heart’s electrical rhythm or conduction. It may feel skipped, irregular, suddenly fast, or slow; an ECG is needed to identify the rhythm.[2][5]

THE MECHANISM, EXPLAINED

Same beat count. Different timing.

Each marker = one beatTime runs left to right
Schematic: evenly spaced
Schematic: unevenly spaced

The same beat count can have different spacing. These are conceptual timing markers without a time scale, not ECG traces or a specific arrhythmia. A rate display cannot diagnose an arrhythmia. [2]

Arrows show functional relationships. This is not an individual diagnostic result.

It is spring. During a week of overtime, Misaki’s coffee intake has risen to five cups a day. In bed one night, she notices an odd pattern: beat, beat, pause, thump. It seems as though a beat has stopped, followed by an unusually strong one.

The word arrhythmia does not mean every rhythm change is dangerous. It does, however, need to be distinguished from normal responses to activity or pain. The feeling alone cannot identify the cause. Here are four familiar rhythm problems and the reasons to have them checked.[2][5]

Premature Beats: Why a Beat Can Feel “Skipped”

An electrical impulse outside the sinus node can trigger a beat earlier than expected. That early contraction may not send a strong enough pulse to the wrist, making it feel as if a beat was missed.[5]

Premature atrial beats occur in healthy people and may go unnoticed. Coffee, tea, alcohol, and some cold medicines can act as triggers for some people. Premature ventricular beats are also common and become more frequent with age. They are often benign without underlying heart disease, but frequent beats or an existing heart condition need assessment.[11][24]

In the story, Misaki returns to two coffees a day and goes to bed earlier; eventually she no longer notices the thump. That fictional improvement does not establish the cause or identify her rhythm. Palpitations accompanied by chest pain can also occur with conditions such as angina or a heart attack: call your local emergency number now.[5]

Atrial Fibrillation: An Irregularly Irregular Pulse

In atrial fibrillation, electrical activity in the atria becomes disorganized, often making the pulse intervals irregular. Blood clots can form in the atria and travel to a brain artery, causing an ischemic stroke.[5]

Using data from about 630,000 Japanese health-checkup participants aged 40 or older in 2003, researchers estimated about 700,000 people with atrial fibrillation in Japan in 2005. Later public information describes an estimated total above one million. These are historical estimates and educational materials, not a measured 2026 count. Atrial fibrillation becomes more common with age.[26][27]

Misaki’s 82-year-old grandmother has no symptoms. Her doctor notices an irregular pulse, and an ECG identifies atrial fibrillation. In Japan, March 9 is promoted as Pulse Day, and March 9–15 as Atrial Fibrillation Week, raising awareness of the condition.[27]

  • Public education materials describe an approximately fivefold higher risk of ischemic stroke with atrial fibrillation. An individual’s risk varies with age and other medical conditions.[28]
  • Strokes caused by clots from the heart can be severe. A 2014 information release reports about 20% mortality and about 40% needing care, including people left bedridden. These population figures do not predict an individual’s outcome.[28]
  • Intermittent atrial fibrillation may cause no symptoms, making ECG confirmation important. Appropriate anticoagulation helps prevent stroke; the same material cites a reduction of about 60%. A clinician must weigh treatment benefits against bleeding risk and other individual factors.[28]

Paroxysmal Supraventricular Tachycardia: A Sudden Start and Stop

A very fast, regular rhythm can begin and end abruptly, like a switch being turned on and off. The source describes rates of 160–220 beats per minute, episodes lasting minutes to hours, and occurrence in younger people. Matching those numbers alone does not diagnose supraventricular tachycardia.[25]

Palpitations may come with breathlessness, chest pain, or lightheadedness, and treatments are available. Many episodes are not immediately life-threatening, but you should not decide on your own that a fast rhythm is safe. A first sudden episode or recurring attacks need medical advice. With chest pain, severe breathlessness, fainting, or similar emergency symptoms, call your local emergency number now.[25]

Bradyarrhythmias: A Rhythm That Is Too Slow

Problems generating or conducting the heart’s electrical signals can slow the pulse. Strong vagal effects can also cause slowing. These possibilities require a different assessment from an asymptomatic slow pulse in someone who trains regularly.[2][5]

A slow pulse with unusual mental fogginess, breathlessness, or feeling faint needs medical evaluation. For fainting, altered consciousness, or severe breathlessness, call your local emergency number now. Do not wait to count a particular low rate or time a pause of several seconds.[1][5]

What Online Measurement Can and Cannot Tell You

HeartScan online measurement estimates heart rate from tiny chest vibrations detected by your phone’s accelerometer, using seismocardiography (SCG). It is not an ECG, which records the heart’s electrical activity. The displayed number cannot diagnose atrial fibrillation or another rhythm disorder, and a usual reading cannot rule one out. A new or recurring irregular pulse needs clinical assessment with an ECG or other appropriate rhythm testing.[5][28]

LOOK CLOSER

From tiny chest movements to a heart rate estimate

Reclined, with the phone on the chestChest motion, magnified

Schematic: motion is exaggerated for visibility. Anatomy, size, and displacement are not to scale.

Tiny chest movements

Mechanical vibrations associated with heartbeats

Current step
Accelerometer

Motion detected by the phone resting on the chest

Signal processing

Estimate beat timing within the motion signal

Heart-rate estimate

Estimate heart rate from intervals between beats

SCG (seismocardiography) uses the phone’s motion sensors, not its camera or a finger/wrist measurement. It detects mechanical vibrations, not the heart’s electrical activity. This is not a measurement or an arrhythmia diagnosis.

1 / 4
Place the phone on your chest

Recline comfortably and follow the measurement tool’s guidance.

Shapes, movement, and timing are exaggerated for explanation. Not measured data or an individual diagnostic result. How SCG works

Note the displayed reading, symptoms, start time, duration, and measurement conditions to tell your clinician. An intermittent arrhythmia may have ended by the time you are examined. For chest pain, severe breathlessness, fainting, or other emergency symptoms, prioritize emergency help over another measurement or completing notes.[1][5][28]

Chapter 8: When to Seek Medical Care

Persistent unexplained changes or an irregular pulse need medical advice. Chest pain, severe breathlessness, fainting, or altered consciousness require emergency help now, regardless of the pulse number.[1][5]

THE MECHANISM, EXPLAINED

Symptoms and persistence determine the next step

Chest pain, severe breathlessness, or fainting?
Yes →

Call your local emergency number nowDo not wait to keep measuring

No emergency symptoms
An unexplained change or irregularity persists?
Yes →

Seek medical adviceShare readings, symptoms, and when it started

No; learning your usual resting rate

Rest and measure under consistent conditionsOnline measurement does not replace diagnosis

Reassess if symptoms appear or worsen, regardless of the reading. [1] [2]

Arrows show functional relationships. This is not an individual diagnostic result.

Many heart rate changes are normal responses, but some need professional assessment. Your usual pulse is a clue, not a test that proves you are safe. Do not delay emergency care to take more measurements or check an app.

Arrange Medical Advice Promptly

The following are reasons to seek advice, not diagnoses. Training and medications can make a low pulse usual for some people. Explain whether the pattern is new and whether you have symptoms.[1][5]

  • A resting pulse that persistently exceeds 100 or remains below 50.[5]
  • A newly irregular rhythm or repeated episodes of irregularity.[5]
  • A sustained change from your usual rate with fatigue or mild dizziness.[1]
  • Resting readings that stay elevated over several days without a clear explanation.[10]
  • Little rise in heart rate with exercise, or unusual symptoms during activity.[10]

Call Your Local Emergency Number Now

Call your local emergency number now for chest pain or pressure, severe breathlessness, fainting, altered consciousness, marked dizziness or feeling about to pass out, or palpitations accompanied by a cold sweat. Do not wait for a pulse above 100, below 50, or a pause lasting a certain number of seconds. A reading within a usual range cannot rule out an emergency.[1][5]

  • A suddenly fast or slow pulse accompanied by chest pain or severe breathlessness.[1]
  • Palpitations with a cold sweat, marked dizziness, or a feeling that you may faint.[5]
  • A slow pulse with fainting, altered consciousness, or severe difficulty breathing.[5]

What to Tell Your Clinician

Routine notes can help an assessment, but never gather them at the expense of emergency care. Even if an episode has stopped, its start time and duration can be valuable because arrhythmias often come and go.[5][28]

  • Your usual pulse and the reading that concerned you.[5]
  • Whether the rhythm was regular or felt as if it skipped.[5]
  • Symptoms such as fatigue, dizziness, breathlessness, or chest pain.[5]
  • When the episode began, how long it lasted, what you were doing, and any medicines you take.[1][28]

Conclusion: A Small Letter From Your Body

Heart rate is neither a countdown of remaining life nor a stand-alone diagnosis. It is a clue to interpret alongside your usual readings, the reason for a change, its duration, rhythm, and symptoms.

THE MECHANISM, EXPLAINED

Measure. Add context. Check symptoms. Act.

  1. Measure consistently

    Rest; keep time, posture, and method comparable

  2. Add context

    Note sleep, activity, fever, medicines, and other influences

  3. Check symptoms

    Look for chest pain, breathlessness, fainting, or persistent change

  4. Choose the next action

    Emergency help for urgent symptoms; advice for persistent change; records for comparison

The number is a starting point. Context and symptoms make it useful for deciding what comes next.

Arrows show functional relationships. This is not an individual diagnostic result.

By autumn, Misaki’s morning pulse has settled around 58. A year earlier, 92 before a presentation, 73 during a cold, or a nighttime thump might all have frightened her. Now she knows that each has possible explanations, but that a number alone cannot establish the cause. She also remembers how an irregular pulse led to her grandmother’s atrial fibrillation diagnosis.

Your pulse is like a small letter telling you something about your current condition. Three ideas help you read it.

The two pumps that have been working since before you were born are still adjusting their rhythm to your body’s needs. An online pulse reading is another clue to consider in that context. Record concerning changes and seek advice; if emergency symptoms occur, stop measuring and get help.

  • The heart has two pumps. Nerves and hormones adjust its pace as stress, fever, activity, and sleep change.[6][7][8]
  • Reference ranges are broad, and comparisons with your own readings under similar conditions can help. A baseline is not a diagnosis.[1][3][4]
  • Look beyond the number to the reason, persistence, rhythm, and symptoms. Seek advice for unexplained changes or irregularity, and call your local emergency number now for chest pain, severe breathlessness, fainting, or other emergency symptoms.[1][5]

Frequently Asked Questions

Is a resting heart rate in the 90s normal?

A resting heart rate in the 90s is within the general adult range of 60–100 beats per minute, but above the 45–85 reference interval used for Japanese checkups taken lying down. These references have different purposes and measurement conditions; neither diagnoses disease by itself. Seek medical advice if readings remain in the 90s after a trigger such as stress has passed, or stay higher than your usual pulse. Chest pain, severe breathlessness, or fainting requires a call to your local emergency number now, even with a pulse in the 90s.[1][2][3][10]

Can a resting heart rate in the 50s be healthy?

A resting heart rate in the 50s can be normal in younger people or those who exercise regularly, and is within the Japanese checkup reference interval. Medicines and heart conditions can also cause a low rate, so lower is not always better. Seek advice for a new change, fatigue, dizziness, or breathlessness. Fainting, severe breathlessness, or chest pain requires a call to your local emergency number now.[1][2][3][5]

How can I lower my heart rate quickly?

If your pulse is temporarily elevated after activity or with nervousness, sitting and resting may allow it to settle. Slow, comfortable breathing may help you feel calmer, but it is not treatment for an arrhythmia. A suddenly very fast pulse or recurring episodes need medical advice; chest pain, severe breathlessness, or fainting means call your local emergency number now. Over time, endurance exercise or yoga can lower resting heart rate on average, but individual responses vary.[9][16][25]

Do women have higher resting heart rates than men?

In a wearable study of about 90,000 people, women had higher average resting heart rates than men across age groups. Differences between individuals were much larger, so sex alone does not determine a healthy rate for you. Compare measurements taken under similar conditions with your own previous readings, and consider any symptoms.[4]

Does resting heart rate change with age?

In a study of about 90,000 people, average resting heart rate rose until around age 50 and then declined, but this is not a pattern every individual must follow. The size of the reduction associated with exercise also varies with age, while premature ventricular beats and atrial fibrillation become more common in older adults. Do not dismiss a new change or irregular rhythm as simply aging; seek medical advice.[4][11][16][27]

Can HeartScan online measurement diagnose an arrhythmia?

No. HeartScan online measurement estimates pulse rate; it is not an ECG and does not diagnose rhythm disorders. A usual reading cannot rule out an arrhythmia. Seek medical advice for persistent unexplained changes or an irregular pulse; an ECG or other appropriate testing may be needed. For chest pain, severe breathlessness, fainting, or other emergency symptoms, call your local emergency number now rather than waiting for another reading.[1][5][28]

Sources

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  2. MSD Manual Consumer Version. Overview of Abnormal Heart Rhythms. 2024.
  3. 日本人間ドック・予防医療学会. 判定区分表に関するQ&A【心臓】.
  4. Quer G, Gouda P, Galarnyk M, Topol EJ, Steinhubl SR. Inter- and intraindividual variability in daily resting heart rate and its associations with age, sex, sleep, BMI, and time of year. PLOS ONE. 2020. doi:10.1371/journal.pone.0227709.
  5. 日本心臓財団. 心拍数と心臓病(健康ハート叢書17). 監修:山科章. 2015.
  6. National Heart, Lung, and Blood Institute. How the Heart Beats. 2025.
  7. MSD Manual Consumer Version. Biology of the Heart. 2025.
  8. Klabunde RE. Regulation of Pacemaker Activity. Cardiovascular Physiology Concepts.
  9. Cleveland Clinic. Sinus Arrhythmia. 2022.
  10. 心リハ♡NOW. Vol.16 たかが脈拍 されど脈拍?!
  11. MSD Manual Consumer Version. Ventricular Premature Beats. 2024.
  12. Karjalainen J, Viitasalo M. Fever and cardiac rhythm. Arch Intern Med. 1986;146(6):1169-71.
  13. Radin JM, Wineinger NE, Topol EJ, Steinhubl SR. Harnessing wearable device data to improve state-level real-time surveillance of influenza-like illness in the USA: a population-based study. Lancet Digit Health. 2020. doi:10.1016/S2589-7500(19)30222-5.
  14. Mishra T, Wang M, Metwally AA, et al. Pre-symptomatic detection of COVID-19 from smartwatch data. Nat Biomed Eng. 2020. doi:10.1038/s41551-020-00640-6.
  15. Radin JM, Quer G, Ramos E, et al. Assessment of prolonged physiological and behavioral changes associated with COVID-19 infection. JAMA Netw Open. 2021;4(7):e2115959.
  16. Reimers AK, Knapp G, Reimers CD. Effects of exercise on the resting heart rate: a systematic review and meta-analysis of interventional studies. J Clin Med. 2018;7(12):503.
  17. D'Souza A, Bucchi A, Johnsen AB, et al. Exercise training reduces resting heart rate via downregulation of the funny channel HCN4. Nat Commun. 2014;5. doi:10.1038/ncomms4775.
  18. Drezner JA, Sharma S, Baggish A, et al. International criteria for electrocardiographic interpretation in athletes: consensus statement. Br J Sports Med. 2017;51:704-31.
  19. Andersen K, Farahmand B, Ahlbom A, et al. Risk of arrhythmias in 52 755 long-distance cross-country skiers: a cohort study. Eur Heart J. 2013. doi:10.1093/eurheartj/eht188.
  20. 本川達雄. ゾウの時間 ネズミの時間. 中公新書. 同書を引用した教材.
  21. Levine HJ. Rest heart rate and life expectancy. J Am Coll Cardiol. 1997;30(4):1104-6.
  22. 更科功. 現代ビジネス. 2019.
  23. Zhang D, Shen X, Qi X. Resting heart rate and all-cause and cardiovascular mortality in the general population: a meta-analysis. CMAJ. 2016;188(3):E53-63.
  24. MSD Manual Consumer Version. Atrial Premature Beats. 2024.
  25. MSD Manual Consumer Version. Paroxysmal Supraventricular Tachycardia (SVT, PSVT). 2024.
  26. 塗木徳人(鹿児島医療センター). 不整脈って? 心臓・血管病市民公開講座資料. 2012. 引用元:Inoue H, et al. Int J Cardiol. 2009;137:102-7.
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About This Article

Source-based, AI-assisted educational content by the HeartScan editorial team. This article has not been medically reviewed. Misaki and her family are fictional, and their experiences are illustrative examples.

This is general educational information, not a substitute for medical diagnosis or treatment. A personal baseline, online measurement, or app reading cannot diagnose or rule out disease. Online measurement estimates pulse rate and is not an ECG. Consult a healthcare professional about concerning changes.